Rowaida Taha | Hamad Bin Khalifa University

Rowaida Taha

Senior Research Associate

Email

rotaha@hbku.edu.qa

Phone

+974 445 42543

Rowaida Taha

Senior Research Associate

Entity

Qatar Biomedical Research Institute

Divison

Neurological Disorders Research Center

Biography

Ms. Rowaida Ziad Taha earned her Bachelor’s degree in Genetic Engineering and Biotechnology from Jordan University of Science and Technology, College of Science (JUST). In 2008, she joined Weill Cornell Medical College, Doha, Qatar, as a Research Technician. Her work was mainly on projects related to stem cell and cancer biology research, those which included a variety of molecular and cellular biology techniques. In 2010, she worked at Shafallah Medical Genetics Center (SMGC) as a Research Assistant focusing on delineating the spectrum and distribution of Family Mediterranean Fever (FMF) among Arab patients and on mutation identification for MEFV, to assist research into the genotype-phenotype correlation of these patients. In 2014, Ms. Taha joined QBRI as a Research Associate. She has mainly contributed to several variation verifications and sequencing validation for genes pertaining to Autism Spectrum Disorders.

  • Family-Based Genome-Wide Association Study of Autism Spectrum Disorder in Middle Eastern Families. Genes (Basel). 2021 May 18;12(5):761. doi: 10.3390/genes12050761.
  • Transcriptome of Tumor-Infiltrating T Cells in Colorectal Cancer Patients Uncovered a Unique Gene Signature in CD4+ T Cells Associated with Poor Disease-Specific Survival. Vaccines (Basel). 2021 Apr 1;9(4):334. doi: 10.3390/vaccines9040334.
  • Differential gene expression of tumor-infiltrating CD33+ myeloid cells in advanced- versus early-stage colorectal cancer. Cancer Immunol Immunother. 2021 Mar;70(3):803-815. doi: 10.1007/s00262-020-02727-0.
  • T-cell responses and therapies against SARS-CoV-2 infection. Immunology. 2021 Jan;162(1):30-43. doi: 10.1111/imm.13262.
  • Expression of immune checkpoints and T cell exhaustion markers in early and advanced stages of colorectal cancer. Cancer Immunol Immunother. 2020 Oct;69(10):1989-1999. doi: 10.1007/s00262-020-02593-w.
  • Differential gene expression of tumor-infiltrating CD4+ T cells in advanced versus early stage colorectal cancer and identification of a gene signature of poor prognosis. Oncoimmunology. 2020 Sep 30;9(1):1825178. doi: 10.1080/2162402X.2020.1825178
  • Transcriptomic Profiling of Circulating HLA-DR- Myeloid Cells, Compared with HLA-DR+ Myeloid Antigen-presenting Cells. Immunol Invest. 2020 Jul 29:1-12. doi: 10.1080/08820139.2020.1795875
  • Differential gene expression of tumor-infiltrating CD33+ myeloid cells in advanced- versus early-stage colorectal cancer. Cancer Immunol Immunother. 2021 Mar;70(3):803-815. doi: 10.1007/s00262-020-02727-0.
  • PD-L1 Blockade by Atezolizumab Downregulates Signaling Pathways Associated with Tumor Growth, Metastasis, and Hypoxia in Human Triple Negative Breast Cancer. Cancers (Basel). 2019 Jul 25;11(8):1050. doi: 10.3390/cancers11081050.

  • Epigenetic modifications in the immune checkpoint genes PD-1, CTLA-4, TIM-3, LAG-3, PD-L1 and TIGIT in human breast tumor tissues (Submitted).

  • Multiplex epithelium dysfunction due to CLDN10 mutation: the HELIX syndrome. Genet Med. 2018 Feb;20(2):190-201.

  • Clin Case Rep. 2017 May 12;5(6):1013-1017.

  • Overlap of familial Mediterranean fever and hyper-IgD syndrome in an Arabic kindred. Journal of Clinical Immunology.;35(3):249-53.

  • Distal trisomy 10q syndrome, report of a patient with duplicated q24.31 - qter, autism spectrum disorder and unusual features. Clin Case Rep.; 2(5):201-5